Monday, April 23, 2012


DIPLOMA IN CHILD HEALTH EXAMINATION ……………………….         2010
Paper – I Applied Basic Medical Sciences in Pediatrics
Time : 3 Hours                                                                                                                   Maximum Marks : 100
Answer ALL questions
  1. Enumerate the Diagnostic approach to short stature                      (20 Marks)
  2.                                                                                                                                                 (10x8=80 marks)
1. Exudative pleural effusion
2. tachyarrythmia
3. management of severe dehydration in acute diarrheal disorder
4.partially treated pyogenic meningitis
5. how will you evaluate  headache in a 12 year old child.
6. list the events in pathogenesis of diabetic ketoacidosis
7. Etiology and clinical features of voiding dysfunction in children
8. Evaluation and diagnosis of appendicitis in children

DIPLOMA IN CHILD HEALTH EXAMINATION ……………………….         2010
Paper – I Applied Basic Medical Sciences in Pediatrics
Time : 3 Hours                                                                                                                   Maximum Marks : 100
Answer ALL questions
  1. Evaluation of hearing impairment in children                    (20 Marks)
  2.                                                                                                                                                 (10x8=80 marks)
  1. Febrile seizures
  2. Fulminant hepatic failure
  3. Glasgow coma scale
  4. Beta Thalassemia
  5. Hepatitis E virus infection
  6. Indications for and complications of renal biopsy
  7. Intensive care unit management of acute severe asthma exacerbation in children
  8. Pathophysiology and etiology of edema in children

DIPLOMA IN CHILD HEALTH EXAMINATION ……………………….         2010
Paper – I Applied Basic Medical Sciences in Pediatrics
Time : 3 Hours                                                                                                                   Maximum Marks : 100
Answer ALL questions
  1. Diagnosis and managemnt of pyogenic meningitis in 3 year old child               (20 Marks)
  2.                                                                                                                                                 (10x8=80 marks)
1. tension pneumothorax
2. Physiology of lactation
3. management of status epilepticus
4. Presentation, diagnosis, and clinical course of vesicoureteral reflux
5. Primary humoral immune deficiencies.
6. Pulmonary hypertension
7. Rapid diagnostic tests for tuberculosis
8. Renin-angiotensin system

TOC www.acepgmed.com 24-04-2012


Topic
URL
ACE PG Concept Bridge (CB) Management of Diarrhea Part 01C
ACE PG Concept Bridge (CB) Management of Diarrhea Part 01B
ACE PG Concept Bridge (CB) Management of Diarrhea Part 01A
ACE PG Concept Bridge (CB) Chest Radiography Interpretation 03
ACE PG Concept Bridge (CB) Chest Radiography Interpretation 02
ACE PG Concept Bridge (CB) Chest Radiography Interpretation 01
ACE PG Concept Bridge (CB) Blood Supply brain
Applied anatomy Thorax-self-Learning module 03
Applied anatomy Thorax-self-Learning module 02
Applied anatomy Thorax-self-Learning module 01
Concept Bridge (CB) 004 Deep neck space infections 01
ACE PG Concept Bridge (CB) 003
ACE Concept Bridge (CB) 002 Clinical Scenarios 02
ACE PG Concept Bridge (CB) 001 Clinical Scenarios 01
CB Series ECG Tutorial 06- Back to Basics
CB Series ECG Tutorial 05- Back to Basics
CB Series ECG Tutorial 04- Back to Basics
CBT Series  ECG Tutorial 03- Back to Basics
CBT Series ECG Tutorial 02- Back to Basics
CBT Series ECG Tutorial 01- Back to Basics.
CBT Cases 003 A case of Acdotic Breathing
Ace PGMed  Change in Video Format
Ace PGMed CPC clinico pathological corelationship case discussion 001
ECG tutorial Basic principles of ECG analysis 001
ECG tutorial Basic principles of ECG analysis 002
ECG tutorial Basic principles of ECG analysis 003
ECG tutorial Basic principles of ECG analysis 004
ECG tutorial Basic principles of ECG analysis 005
ECG tutorial Basic principles of ECG analysis 006
ECG tutorial Basic principles of ECG analysis 007
ECG tutorial Basic principles of ECG analysis 008
Giant A Wave
Small for gestational age infant 001
Small for gestational age infant 002
ECG tutorial Basic principles of ECG analysis Pediatric ECG 001
ECG tutorial Basic principles of ECG analysis Pediatric ECG 002
ECG tutorial Basic principles of ECG analysis Pediatric ECG 003
ECG tutorial Basic principles of ECG analysis Pediatric ECG 004
ECG tutorial Basic principles of ECG analysis Pediatric ECG 005
ECG tutorial Basic principles of ECG analysis Pediatric ECG 006
ECG tutorial Basic principles of ECG analysis Pediatric ECG 007
Cases 002  Two cases of Dyspnoea.
CBT Cases 001 A case of Sudden onset Dyspnea
Introduction and how to use Ace PG Med
PG Entrance Test Practice Session (PGET) 011
http://youtu.be/BxX8Fnk6pm4
PG Entrance Test Practice Session (PGET) 010
http://youtu.be/cIknJYcqTKM
PG Entrance Test Practice Session (PGET) 009
http://youtu.be/rang52TCuvQ
PG Entrance Test Practice Session (PGET) 008
PG Entrance Test Practice Session (PGET) 007
PG Entrance Test Practice Session (PGET) 006
ACE PG Med PG Entrance Test Practice Session (PGET) 005
ACE PG Med PG Entrance Test Practice Session (PGET) 004
ACE PG Med PG Entrance Test Practice Session (PGET) 003
PG Entrance Test Practice Session (PGET) 002
PG Entrance Test Practice Session (PGET) 001
PG Entrance Test Practice Session  001 Cardiology
PG Entrance Test Practice Session 002 Cardiology
PG Entrance Test Practice Session 003 Cardiology
PG Entrance Test Practice Session 004 Cardiology
PG Entrance Test Practice Session 005 Cardiology
PG Entrance Test Practice Session 006 Cardiology
PG Entrance Test Practice Session 007 Cardiology
PG Entrance Test Practice Session Surgery 001
PG Entrance Test Practice Session Surgery 002
PG Entrance Test Practice Session Surgery 003
PG Entrance Test Practice Session Surgery 004
PG Entrance Test Practice Session Surgery 005
PG Entrance Test Practice Session Surgery 006

Pediatric bipolar disorder (PBD) (Pediatrics in Review Vol.32 No.11 November 2011, DOI: 10.1542/pir.32-11-502)
The clinical presentation of PBD often does not meet the Diagnostic and Statistical Manual of Mental Disorders criteria for bipolar disorder (BD), which was developed in adults and not adapted for children. The diagnosis becomes difficult because symptom presentation is variable and largely dependent on developmental age. Although discrete episodes of mania and depression that define BD in adults may match the presentation of some patients who have BD in adolescence, patients who have BD of childhood and prepubertal onset may or may not manifest these clear-cut episodes. The duration of episodes in children may be as short as 1 to 2 days. Mood symptoms can be chronic, can present as predominantly mixed episodes, or can continuously cycle rapidly, with severe irritability or aggression as the usual presenting symptoms. Disruptive behavior, hyperarousal,racing thoughts, elation, and grandiosity also may characterize the moods. A major depressive presentation is associated with a significant risk for developing subsequent BD. Poor psychosocial skills as well as cognitive and attention deficits may manifest along with mood and behavior changes. The chronic relapsing mode of PBD may extend into early adulthood. Complicating the diagnosis of PBD is the high rate of comorbidity with attention deficit hyperactivity disorder (ADHD) and psychiatric disorders such as anxiety, conduct, substance abuse, and oppositional defiant disorders. Research has suggested that physical, sexual, and emotional childhood traumas that occur at a time of sensitivity of the maturing central nervous system may predispose to and modulate the clinical expression and course of the PBD

Update in Surfactant Therapy(NeoReviews 2011;12;e625-e634, DOI: 10.1542/neo.12-11-e625)
Evidence for Use of Exogenous Surfactant in Neonatal Lung Diseases Other Than RDS

Meconium Aspiration
Meconium inactivates components of surfactant, thereby reducing the effectiveness of endogenous surfactant. Airway obstruction, inflammation, protein leak, and retained fetal lung fluid all contribute to the pathogenesis of meconium aspiration syndrome
The potential advantages to providing exogenous surfactant therapy include restoration of alveolar surfactant with the potential for improved distribution owing to retained fetal lung fluid and alveolar edema fluid as discussed above. However, the clinical trials to date only demonstrate a reduction in the need for ECMO

Congenital Diaphragmatic Hernia
CDH is characterized by pulmonary hypoplasia, poor lung compliance and likely altered numbers of alveolar type 1 and 2
epithelial cells.
In CDH lung as maldeveloped, rather than immature. The severe respiratory compromise associated with CDH is exacerbated by left ventricular and pulmonary arterial hypoplasia, arterial intimal remodeling, and pulmonary ypertension,
as well as altered alveolar epithelial cell populations. This more complex description of CDH pathophysiology and the variable timing of surfactant administration postnatally may help to explain why surfactant replacement has not shown a consistent benefit to date
Bronchiolitis
RSV binds to Toll-like receptors on epithelial cells lining the upper and lower airways and initiates a brisk inflammatory response that contributes to epithelial cell injury and reduced surfactant production. Epithelial cell dysfunction, inflammatory cytokines, and epithelial barrier injury lead to alveolar edema and contribute to surfactant abnormalities and respiratory failure in infants with bronchiolitis. The evidence base supporting the widespread use of surfactant for bronchiolitis is insufficient at this time and additional randomized controlled trials are warranted.
Genetic Disorders of the Surfactant System
They are mutations in SP-B, SP-C, and ABCA3.
They present as Neonatal respiratory distress occurring in term and late preterm infants with no other risk factors for respiratory disease
Infants with genetic disorders of the surfactant system are often indistinguishable from late preterm infants with RDS or from term infants with surfactant dysfunction due to neonatal pneumonia. Unless the treating physician is aware of a prenatal diagnosis or family history of one of these genetic disorders, it is understandable that these infants will initially be treated with surfactant due to their clinical presentation. However, there is no evidence base supporting the continued
use of surfactant in these patients